تريخی

د ويکيپېډيا لخوا

همدا مخ د ژباړلو لپاره نومول شوی
ددې ليکنې په ژباړلو کې د پښتو ويکيپېډيا سره ستاسو مرستې ته اړ يو. يواځې هغه کارونکي چې په همدې ژبې لاسبری لري، کولای شي دا مخ پښتو ته واړوي. که چېرته تاسو د دغه مخ د ژباړې اړونده لارښوونې او يا هم کوم وړانديزونه لری، نو مهرباني وکړی د همدې مخ د خبرو اترو په برخه کې خپلې څرګندونې وليکی.

د ژباړې لپاره نومېدلي، نور مخونو دلته وګورۍ.

تريخی Gallbladder

تريخی #5 دی

لاطين	vesica biliaris
Gray's	subject #250 1197
Artery	Cystic artery
Vein	Cystic vein
وژی	celiac ganglia, vagus^[1]
Precursor	Foregut
MeSH	Gallbladder
Dorlands/Elsevier	g_01/12383343

تريخی (او يا هم چې په انګليسي ژبه cholecyst يا کله ناکله gall bladder بلل کېږي.) د انسان په بدن کې يو ناک-وزمه غړی دی چې د ځيګر د شاه نه مخ په ښکته زوړند دی او نژدې 50 ml صفرا يا ترخه چې په انګليسي يې bile (يا "gall") بولي، تر هغه مهاله پورې ساتي ترڅو چې بدن ته د خواړو د هاضمې په مهال پکار راځي.

[سمول] اناتومي

Biliary tract

تريخی د انسان په بدن کې نژدې 7-10 cm اوږدوالی لري او په همدې توګه تريخی ګاټه زرغون رنګ لري چې همدا رنګ يې د ترخې يا صفرا د شتون په سبب اخيستی دی، نه د دغه ژواندي يا اورګان د جوړښت په سبب. تريخی د انسان په خېټه کې ځيګر او ډيوډونم پورې د ترخې يا صفرا د نلکې په مرسته نښتې کڅوړه ده.

The cystic duct connects the gallbladder to the common hepatic duct to form the common bile duct.

The common bile duct then joins the pancreatic duct, and enters through the hepatopancreatic ampulla at the major duodenal papilla.

[سمول] مايکروسکوپي اناتومي

The different layers of the gallbladder are as follows:

The gallbladder has a simple columnar epithelial lining characterized by recesses called Aschoff's recesses, which are pouches inside the lining.
Under the epithelium there is a layer of connective tissue (lamina propria).
Beneath the connective tissue is a wall of smooth muscle (muscularis muscosa) that contracts in response to cholecystokinin, a peptide hormone secreted by the duodenum.
There is essentially no submucosa separating the connective tissue from serosa and adventitia.

Stained section of a gall bladder showing the highly convoluted mucosal folds

[سمول] کړنې

The gallbladder stores about 50 ml of bile (1.7 US fluid ounces / 1.8 Imperial fluid ounces), which is released when food containing fat enters the digestive tract, stimulating the secretion of cholecystokinin (CCK). The bile, produced in the liver, emulsifies fats and neutralizes acids in partly digested food.

After being stored in the gallbladder, the bile becomes more concentrated than when it left the liver, increasing its potency and intensifying its effect on fats. Most digestion occurs in the duodenum.

[سمول] په ناروغيو کې د تريخي رول

Cholestasis is the blockage in the supply of bile into the digestive tract. It can be "intrahepatic" (the obstruction is in the liver) or "extrahepatic" (outside the liver). It can lead to jaundice, and is identified by the presence of elevated bilirubin level that is mainly conjugated.

Biliary colic is when a gallstone blocks either the common bile duct or the duct leading into it from the gallbladder.

Up to 25% of all people have gallstones (cholelithiasis), composed of lecithin and bile acids. These can cause abdominal pain, usually in relation with the meal, as the gallbladder contracts and gallstones pass through the bile duct.

Acute or chronic inflammation of the gallbladder (cholecystitis) causes abdominal pain. 90% of cases of acute cholecystitis are caused by the presence of gallstones. The actual inflammation is due to secondary infection with bacteria of an obstructed gallbladder, with the obstruction caused by the gallstone.

When gallstones obstruct the common bile duct (choledocholithiasis), the patient develops jaundice and liver cell damage. It is a medical emergency, requiring endoscopic or surgical treatment such as a cholecystectomy.

A rare clinical entity is ileus (bowel) obstruction by a large gallstone, or gallstone ileus. This condition develops in patients with longstanding gallstone disease, in which the gallbladder forms a fistula with the digestive tract. Large stones pass into the bowel, and generally block the gut at the level of Treitz' ligament or the ileocecal valve, two narrow points in the digestive tract. The treatment is surgical.

Cancer of the gallbladder is a rare but highly fatal disease. It has been associated with gallstone disease, estrogens, cigarette smoking, alcohol consumption and obesity. Despite aggressive modern surgical approaches, advanced imaging techniques, and endoscopy, nearly 90% of patients die from advanced stages of the disease and experience pain, jaundice, weight loss, and ascites.

Polyps (growths) are sometimes detected during diagnostic tests for gallbladder disease. Small gallbladder polyps (up to 10 mm) pose little or no risk, but large ones (greater than 15 mm) pose some risk for cancer, so the gallbladder should be removed. Patients with polyps 10 mm to 15 mm have a lower risk but they should still discuss removal of their gallbladder with their physician. Of special note is a condition called primary sclerosing cholangitis, which causes inflammation and scarring in the bile duct. It is associated with a lifetime risk of 7% to 12% for gallbladder cancer. The cause is unknown, although primary sclerosing cholangitis tends to strike younger men who have ulcerative colitis. Polyps are often detected in this condition and have a very high likelihood of malignancy.